Two strains of the carbapenem-resistant KP sequence type 258 (ST258), KP35, and NJST258_2 (Ahn et al., 2016; Deleo et al., 2014), induced higher levels of TNF-α, IL-1β, CXCL10, and type I IFNs in sarm1−/− macrophages than in wild-type cells (Figure S1), demonstrating that SARM1 contribution to KP-triggered inflammatory responses is observed following infection with multidrug resistant strains. The gene discussed is CXCL10; the disease is infection.