Interestingly, we found that knockdown of c‐Met, but not Axl partially restored the proliferative capacity of H460 βIIISH4 cells following MP‐470 treatment compared to siGFP knockdown cells (Figure 6B), suggesting that the expression of c‐Met, but not Axl, is playing an important role in mediating the anti‐proliferative effect of MP‐470 activity in our cell models of NSCLC (See Figure S5 for detailed schematic of proposed model). Here, AXL is linked to non-small cell lung carcinoma.