MYC and thyroid gland carcinoma: The mechanism may be that lncRNAMALAT1 binds to miR-204 to increase the expression of IGF2B2, which can recognize the M6A modification of MYC to increase the expression of MYC, and finally accelerate growth and invasiveness, thus promoting the progression and metastasis of thyroid cancer, suggesting that lncRNAMALAT1 can be adopted as a hopeful molecular regulator of thyroid carcinoma [11].