It is important to note that malnutrition of other dietary components in patients with PEI or post-PD may also be attributable to the development of hepatic steatosis, as evidence of increased levels of serum taurine and decreased methionine, tyrosine, albumin, cholinesterase, zinc, and total serum cholesterol levels was reported in such patients [13]. This evidence concerns the gene BCHE and Hepatic steatosis.