Mitochondrial dysfunction is an important part of apoptosis caused by MI and is related to the impaired permeability of the mitochondrial outer membrane, causing the release of apoptosis-related proteins, such as apoptosis-inducing factor, Bcl-2 proteins, and cytochrome c (Ong and Gustafsson, 2012; Del Re et al., 2019). This evidence concerns the gene FAM215A and myocardial infarction.