Moreover, the expression of miR-200c can be inhibited by binding to miR-200c binding sites to further promote the growth of breast cancer cells, and the expression levels of miR-200c target genes ZEB1 and ZEB2 can be increased, while the highly expressed ZEB1 and ZEB2 can promote the epithelial-mesenchymal transition of cancer cells. Here, ZEB2 is linked to breast carcinoma.