IL17A and Sepsis: Furthermore, prolonged transient neonatal antibiotic exposure suppresses IL-17A-producing ILC3 during the first 2 weeks of life, promoting bacterial translocation and increasing susceptibility to K. pneumoniae-induced sepsis, and recolonization with mature microbiome raises the level of IL-17A-producing ILC3 in mice, reducing their susceptibility to late-onset sepsis (LOS) (83).