Given that the differential expression of Cxcl9/10/11 is the highest between GFP and Bic tumor cells and CXCL9/10/11 share similar regulatory mechanisms and bind to the same receptor, CXCR3 (22), we chose CXCL9 as the representative to investigate how breast cancer cell–derived miR-155 promotes T cell recruitment via upregulating this chemoattractant. Here, CXCR3 is linked to neoplasm.