As expected, the Bic-CM was more potent in attracting activated T cells than GFP-CM, and T cell migration toward GFP-CM and Bic-CM was significantly attenuated and the difference diminished by CXCR3 blockade (Figure 5, G and H), suggesting the CXCL9/10/11-CXCR3 axis plays an essential role in tumor-intrinsic miR-155–mediated T cell influx to the tumor. The gene discussed is CXCL9; the disease is neoplasm.