In vivo, the consequence of a CBS/H2S-mediated pseudohypoxia in DS can include impairments in neuronal metabolic activity, neurotransmission, neuronal electrical activity generation, and neurobehavioral function and memory generation; according to recent studies, these alterations can also be reversed by genetic correction or pharmacological inhibition of CBS [[7], [8], [9]]. The gene discussed is CBS; the disease is Dravet syndrome.