The result in Figure 7 showed that the expressions of MYD88, p–NF–κBp65/NF-κBp65, TLR4, and TLR2 in the COPD group were significantly higher than those of the control group (P < 0.01), and the expression of p-IκBa/IκBa was remarkably lower than that of the control group (P < 0.01). This evidence concerns the gene TLR2 and chronic obstructive pulmonary disease.