KRCC1 and infection: Although we cannot conclude that upregulation of prsA2 mRNA detected by RNA-seq from ΔsigL strain would function as a chaperone protein or as a regulatory RNA to hly mRNA during infection (Ignatov et al., 2020), this finding suggests the interplay between PrsA2 and PrsA1 expression levels in the presence or absence of SigL.