Similarly, the results of our analysis suggest that the lncRNA, KCNQ1OT1, acts as a competitive endogenous RNA that competitively binds tumor-suppressive miR-378a-3p, resulting in increased expression of RBMS1 within tumors via the KCNQ1OT1/miR-378a-3p/RBMS1 axis. The gene discussed is KCNQ1OT1; the disease is neoplasm.