AKT1 and Duchenne muscular dystrophy: Moreover, De palma et al. state that, in Duchenne Muscular Dystrophy, the treatment of abnormal autophagy pathways such as AKT (Protein Kinase B) and mTOR (mammalian target of rapamycin) results in significantly reduced muscle inflammation and fibrosis questioning whether this pathway may also be an important pharmacological target [24].