We found significantly more Ctip2/EdU double-positive cells and significantly fewer Satb2/EdU and Brn2/EdU double-positive cells in the IUGR fetal brains (Figures 2B,C), suggesting delayed transitioning of fetal NSCs from producing early-born (Ctip2-positive) to late-born (Satb2-positive and Brn2-positive) neurons. The gene discussed is SATB2; the disease is fetal growth restriction.