Moreover, stromal activation pathways including epithelial–mesenchymal transition, angiogenesis, and TGF-β signaling, as well as immune activation pathways including allograft rejection, interferon γ response, IL2-STAT5 signaling, complement, inflammatory response, and IL6-JAK-STAT3 signaling displayed significant upregulation in ferroptosis subtype B. Tumor-infiltrating immune cells were then quantified. The gene discussed is TGFB1; the disease is neoplasm.