Using promotor reporting assay in neuroblastoma N2a cells with comparing CLOCK:BMAL1 co-expression to non-supplemented basal condition, we found Camk2d promotor was most responsive to CLOCK:BMAL1 activation with a 6.8-fold increase in Camk2d:Luc expression (Supplementary Fig. 8a), and the induction was not due to dexamethasone synchronization in the test (Supplementary Fig. 8b). Here, CAMK2D is linked to neuroblastoma.