KEAP1 and pulmonary fibrosis: The proposed research work shows that autophagy can alleviate the occurrence of pulmonary fibrosis by activating the Keap1/Nrf2 signaling pathway, and autophagy activators can probably effectively slow down lung injury from BLM-induced pulmonary fibrosis mice, as well as increase the apoptosis of lung fibrotic cells and inhibit the proliferation of lung fibrotic cells, while autophagy inhibitors produce the opposite result.