ADAR1 post-transcriptionally increased the abundance of focal adhesion kinase (FAK) protein by binding and editing to a specific intronic site on chr8: 141,702,274 in FAK transcript, resulting in the increased stabilization of FAK mRNA, thereby promoting mesenchymal properties, migration, and invasion of lung adenocarcinoma (Amin et al., 2017). The gene discussed is PTK2; the disease is lung adenocarcinoma.