With similar analyses, future studies may discern how these behavioral endophenotypes may be affected in mutants with graded variations in Cacng2 expression (Stargazer-3 J mice, Waggler) or in mutants with etiologically distinct syndromes of ataxia and absence seizures (lethargic [Cacnb4], ducky [Cacna2d2], tottering [Cacna1a])56. This evidence concerns the gene CACNA1A and cerebellar ataxia.