To further explore the involvement of VAV1ΔC in these tumours, we crossed the Vav1ΔC/ΔC mice with a mouse strain that can express one of the oncogenic versions of K‐RAS (G12D) in a Cre recombinase dependent manner (referred hereafter as LSL‐KrasG12D/+). The gene discussed is KRAS; the disease is neoplasm.