IFNG and gastric carcinoma: It should be mentioned that this model not only explains the development of the diseases described here, but also others, such as EBV-associated gastric carcinoma, where the formation of ectopic lymphoid structures in the gastric mucosa in response to infection by Helicobacter pylori or another inflammatory process causes the infiltration of B cells with EBV latency in the lymphoid aggregates, and the subsequent infection of epithelial cells when expressing MHC-II, due to an increase in IFN-γ [282,283,284,285,286].