CRP and thrombophilia: The mechanisms which lead to the increase in thromboembolic events vary and include endothelial disfunction, cytokine storm, an increase in prothrombotic markers such as D-dimers and fibrinogen, as well as the rise of pro-inflammatory molecules such as interleukin-6 and C Reactive Protein (CRP), which would result in a hypercoagulability state [7].