In addition, the results of this study also suggest that higher Ki-67 meningiomas were more likely to present intratumoral necrosis (p = 0.078) and heterogeneous enhancement (p = 0.037), which corroborated earlier findings that necrosis and “fluid-secreting” low-grade neoplasm were strong predictors of meningioma progression [22,23,24]. This evidence concerns the gene MKI67 and meningioma.