Anchored by glycosyl phosphatidylinositol, CD24 interacts with sialic acid, binding immunoglobulin-like agglutinin-10 (SIGlec-10) to β2 M, which is overexpressed in some tumor tissues and binds to LILRB1 on macrophages to inhibit phagocytosis, resulting in a loss of immune surveillance [53,54]. This evidence concerns the gene LILRB1 and neoplasm.