NFATC1 and pancreatic neoplasm: Previous studies showed that IRF5 and IRF8 are exclusively expressed in spinal microglia and mediate nerve injury‐induced microglial gene expression.[10] c‐MYC has been implicated in the regulation of a variety of biological processes, including growth, differentiation, apoptosis, DNA repair, and protein synthesis.[42] Evidence showed that NFAT2 binds to the promoter of c‐Myc to accelerate pancreatic cancer cell proliferation.[43] Here, we identified five important binding sites of NFAT1 with c‐Myc promoter, and NFAT1 was colocalized with c‐MYC in spinal microglia.