High levels of c-Jun are found in multiple types of cancer [31], and c-Jun overexpression might lead to an overexpression of c-Myc, since in other types of tumors it has been shown that c-Jun controls the exposure of c-Myc binding directly to its promoter, and that induces an overexpression of c-Jun accelerating the c-Myc promoter activity [32]. The gene discussed is MYC; the disease is cancer.