Additional inhibitory receptors such as Natural Killer group 2 member A (NKG2A) [108], TIM-3 [109], T cell immunoglobulin and ITIM domain (TIGIT) [110] and Lymphocyte-activation gene 3 (LAG-3) [111] have been hypothesized to be responsible for T-cell exhaustion in COVID-19. This evidence concerns the gene LAG3 and COVID-19.