Moreover, taking into account the potential role of this cytokine in the process of thyroid tissue damage in HT (similarly to INF-γ, as mentioned above) [36,48,53], as well as the stimulation of virgin T lymphocytes to differentiate into Th17 cells and their secretion of IL-17, which has also been associated with the pathogenesis of this disease [42,51,52], the observed change is not beneficial to the patient. This evidence concerns the gene IL17A and hematocrit.