NFKB1 and colitis: Using macrophage-specific AMPKβ1-deficient mice, Banskota et al. demonstrated that AMPKβ1 deletion could promote nuclear factor-κB (NF-κB) nuclear translocation, thus upregulating the production of proinflammatory cytokines and exacerbating the severity of dextran sodium sulfate (DSS)-induced colitis in mice [21].