PRKN and non-alcoholic fatty liver: Shao et al. showed that the levels of mitophagy-related proteins Parkin and BNIP3L were significantly reduced in non-alcoholic fatty liver model mice, while exenatide intervention increased the expression of Parkin and BNIP3L and the number of autophagosomes, thereby attenuating oxidative stress damage and protecting against non-alcoholic fatty liver and diabetic liver damage [10].