It has been well-established that overexpression of STK17B has closely associated with the tumorigenesis of hepatocellular carcinoma (HCC) and breast cancer [10], and inhibition of STK17B inhibited HCC tumorigenesis and metastasis via AKT/GSK3β/Snail signaling [11]. This evidence concerns the gene SNAI1 and hepatocellular carcinoma.