Patients with ALI exhibited higher levels of proinflammatory cytokines (TNF-α, IL-1β, and IL-6), lower levels of anti-inflammatory cytokines (IL-10), and lower activities of antioxidative enzymes (total antioxidant capacity (T-AOC), superoxide dismutase (SOD), glutathione peroxidase (GSH-Px), and catalase (CAT)) [4]. The gene discussed is TNF; the disease is acute respiratory distress syndrome.