In another aspect, overexpressed pro-oncogenic kinase, protein kinase Cε(PKCε), and phosphatase and tensin homolog (PTEN) loss would synergically activate the NF-κB pathway [42], and the NF-κB pathway was related to the migration and survival of PCa [43,44]. The gene discussed is NFKB1; the disease is posterior cortical atrophy.