PROS1 and hyperinsulinemic hypoglycemia, familial, 4: A total of 8% of individuals did not display any abnormality and thus were not counted for the final evaluation; 12% showed one abnormality and 88% percent presented two to five co-existing abnormalities; 48% of patients had SPS, 24% aPCR phenotype, 11% the FV Leiden mutation, 24% antiphospholipid antibodies, 9% protein S deficiency, and 13% protein C deficiency; 24% patients presented the HR2 haplotype and only one patient presented the Hong Kong mutation, supporting the results of the previous study [11].