The same lung cancer cell line, A549, was used to assess the effect of AA on the epithelial–mesenchymal transition (EMT) induced by the transforming growth factor-β1 (TGF-β1) [50]; it was found that AA inhibited both the viability and the migration ability of A549 cells treated with TGF-β1. This evidence concerns the gene TGFB1 and lung cancer.