The inhibition of IL-1β together with IL-6, alanine aminotransferase, and blood urea nitrogen by AA was also reported in sepsis, another inflammatory disease in which the Notch signaling pathway plays an essential role [77]; as a result, AA was able to diminish sepsis-associated liver, lung, and kidney damage, thus improving survival in experimental animals. The gene discussed is IL1B; the disease is Sepsis.