ODAD2 and chronic rhinosinusitis: ARMC4 pathogenic variants also caused abnormal ODA docking, but in this case, it is more prominent in the distal ciliary axoneme, and it is associated with left-right laterality defects; similarly, in our study, the individual that presented the pathogenic variant had dextrocardia along with bronchiectasis and chronic rhinosinusitis [3,38].