APOA2 and ST Elevation Myocardial Infarction: The observed negative correlation between HDL-apoA-II (but not of its subfractions HDL2-apoA-II and HDL3-apoA-II) and inflammatory markers (CRP and IL-6) in the present study might reflect the diminishing effect of inflammation-driven reduction in HDL-apoA-II availability, thus contrasting the previously established positive association between plasma levels of HDL particles containing apoA-II and inflammatory response in patients with ST-elevation myocardial infarction [21].