MCL-1 can be induced by several signaling events, including FGFR activation [29], and it has been recently described as a hub molecule at the crossroad between cell survival and invasion, able to contribute to the actin cytoskeleton remodeling via SRC in several cancers [30], including triple-negative breast cancer [30] and PDAC [31]. Here, MCL1 is linked to triple-negative breast carcinoma.