IL23A and atopic eczema: Thus, eczematous eruptions associated with IL-23p19 inhibitors could also be explained by the concept of a Th1/Th17 to Th2 switch, i.e., targeting the Th1/Th17 axis could provoke a shift towards Th2-driven immune responses, and, thus, lead to flares of atopic eczema [9,76].