Together with these effects, high levels of IL-17 were detected in a number of inflammatory conditions in the CNS, including cancer [61], which suggests that CCL2 and IL-17 are involved in regulation of the whole-brain response to post-surgical and tumor-development conditions and might be involved in compensatory neuronal network remodeling after brain damage caused by surgical intervention and tumor regrowth. The gene discussed is CCL2; the disease is cancer.