BCL2 and acute myeloid leukemia: Inhibition of TIFA phosphorylation by Aurora inhibitor VX-680 reduced AML cell growth through downregulation of NF-κB mediated anti-apoptotic genes (i.e., BCL-2, BCL-XL) and inflammatory cytokines and chemokines (i.e., C-X-C Motif Chemokine Ligand 1 (CXCL1), Interleukin-8 (IL-8)), leading to apoptosis of AML cells and increased sensibility to chemotherapy [23,171].