As a result of LPS stimulation, genes that were expressed similarly in both the IBD enteroids and colonoids were shown to be involved in the metabolism of porphyrin and ferroptosis (CP), ABC transporters, phagosome, antigen processing and presentation, human cytomegalovirus infection, Herpes simplex virus 1 infection, Epstein–Barr virus infection, human immunodeficiency virus 1 infection, primary immunodeficiency (TAP2/ABCB3), arachidonic acid, glutathione metabolism (GPX1), and nucleocytoplasmic transport, legionellosis, and leishmaniasis (EEF1A1) (Table 15). This evidence concerns the gene GPX1 and inflammatory bowel disease.