Alteration of AUBPs expression/activity contributes to deregulated expression patterns of genes involved in inflammatory (e.g., PTGS2, TNFA) [31,36,37], metabolic (e.g., FGF21, PTEN) [38,39] and carcinogenic processes (oncogenes/tumor suppressors, e.g., MYC, P53) [40,41] and thus contributes to the development of a wide range of chronic metabolic/inflammatory diseases and cancers [9,29]. This evidence concerns the gene MYC and cancer.