APRIL and BAFF also promoted cell growth and survival when binding with BCMA and the transmembrane and cyclophilin ligand activators and calcium modulator (TACI), which further activated the NFκB pathways and increased the expression of antiapoptotic proteins (Mcl-1, Bcl-xL, and Bcl-2) that protect the MM cells from serum-deprivation-induced cell death and dexamethasone. This evidence concerns the gene TNFRSF17 and Miyoshi myopathy.