FPR1 and colorectal carcinoma: Unpublished results from the authors’ group also indicate a crucial anti-angiogenic function of SPMs in CRC: the activation of FPR1 mediated by the probiotic Lactobacillus Rhamnosus (LGG) or specific bacterial products in CRC cells caused an increase of pro-resolving mediators (RvD1 and LXB4) and the consequent inhibition of angiogenesis [51].