Genetic abnormalities causing deficiency of the G6PD enzyme lead to uncontrolled premature hemolysis of RBCs triggered by viral or bacterial infections, sulpha-containing drugs, and certain types of food, manifested mainly as fatigue, pallidness, jaundice, shortness of breath, tachycardia, dark urine, and splenomegaly [3]. The gene discussed is G6PD; the disease is bacterial infectious disease.