OPRM1 and diabetic neuropathy: Our findings are consistent with the reversion of the antinociceptive actions of inhaled H2S with the administration of MOR antagonists in rats with diabetic neuropathy [36], and with the blockade of the analgesia induced by Na2S, another H2S releaser, with pre-treatment with selective antisense oligodeoxynucleotide probes against DOR and MOR [37].