CAT and myocardial infarction: It has also been suggested that oxidative stress produced by free radicals (including its type ROS), as evidenced by a marked increase in the production of lipid peroxidative products associated with decreased levels of antioxidants’ defense system, such as superoxide dismutase (SOD), catalase (CAT) and reduced glutathione peroxidase (GPx), plays a major role in myocardial damage during MI [8].