Therefore, FadA inhibits the function of the 11-AA inhibitory peptide by binding to the 11-AA inhibitory peptide and activates the β-catenin signaling pathway, resulting in an increased expression of transcription factors, oncogenes, Wnt genes, and inflammatory genes, and in the up-regulation of the NF-κB and Wnt pathways, promoting the proliferation of CRC cells. The gene discussed is NFKB1; the disease is colorectal carcinoma.