Similar results were shown in models of acute lung injury after sepsis (decreased TNF-α, IL-1β, and IL-6 secretion) (38, 39) and paraformaldehyde injury (decreased cell death, MyeloPeroxidase activity, parenchymal vascular permeability, TNF-α and IL-6 secretion, and increased IL-10 secretion) (15, 40). This evidence concerns the gene IL10 and Sepsis.